Mr. Leeroy Facebook ad: “Discover The Power of Natto Today!”

Running for 12 days since September 24, 2026. Crush last saw it on October 6, 2026.
Run by Mr. Leeroy on Facebook. Crush is not the advertiser and does not verify its claims. See this ad in Meta's Ad Library(opens in a new tab)
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About this ad
- Meta Ad Library ID
- 1069476489205786
- Platforms
- Facebook, Instagram and Threads
- Relaunches
- 0
Ad text
If you were put on tamsulosin for an enlarged prostate and one day nothing came out anymore — nobody warned you, and there is a reason nobody warned you. I'm a cardiologist. Twenty-nine years. I have spent my entire career looking at one thing: what silts up arteries and how to clear it. For most of those years, if you had asked me about a prostate, I would have told you it wasn't my department. Then my father called me. He was 68. He'd been getting up three, four times a night for two years and told nobody, because his doctor had said it was normal for his age and he took that as a verdict. Eventually the stream got bad enough that he went back. He walked out with tamsulosin. It worked — within a week he was sleeping, and he was grateful. Four months later he called me and asked me a question I could hear him rehearsing. He wanted to know if it was normal that nothing came out anymore. I looked it up while he was still on the phone. Retrograde ejaculation. Semen going backwards into the bladder instead of out. It is not rare. It is not a surprise to anyone who prescribes this drug. It is printed on the label. He had been taking it for four months and he had never heard the phrase. Here is what tamsulosin actually does. It is an alpha-1A blocker. There are receptors in the smooth muscle around your bladder neck, and the drug switches them off so the muscle relaxes and urine can get past your prostate. That's the whole mechanism. It works, and for a man who cannot sleep, that matters. But those exact same receptors do a second job. They snap the internal sphincter shut at the moment of ejaculation, so semen goes out instead of back. Switch them off for the first job and you have switched them off for the second one. There is no way to have one without the other. That is not a side effect in the sense of something unlucky and unexpected. That is the drug doing exactly what it was designed to do, in a place nobody mentioned to you. But that is not the part that made me angry. The part that made me angry is that the drug was never aimed at the disease in the first place. I went and read the urology literature the way a cardiologist reads it — looking for the blood supply. And it is all there, and it has been there for twenty years. Your prostate is fed by microscopic arteries. In men with an enlarged prostate, blood flow through those arteries is measurably reduced. Not as a consequence — as a cause. Chronically starved tissue inflames. Inflamed tissue swells and stiffens. A swollen prostate squeezes the urethra shut. That is the disease, and it starts in the plumbing, not in the gland. Your prostate was never the enemy. At normal size it does its job and causes you no trouble at all. It only becomes a problem when the vessels feeding it start choking shut. And if you think that sounds like fringe theory, ask yourself why there is a hospital procedure called prostatic artery embolization — where an interventional radiologist goes in through your wrist and works on the arteries feeding your prostate. There are clinics advertising it right now. Nobody performs an artery procedure on an organ that has nothing to do with arteries. So what silts up an artery? Fibrin. It is the mesh your body lays down to stop bleeding, and it is the scaffold that plaque builds on. Your body clears it with an enzyme called plasmin. And somewhere after fifty, you stop producing plasmin at the rate you used to. The mesh goes in faster than it comes out. In the smallest vessels in your body, that shows up first. Which is why the earliest warning you will ever get is not chest pain. It is a weak stream. And then it is nothing coming out. That is not the disease. That is the alarm. It is wired to the smallest room in the house, which is why it goes off first — and every man I know has been told to ignore it. Now look at what he was given. Tamsulosin relaxes a muscle around a blockage it does not touch. Saw palmetto works on the swelling and never goes near the vessels. Cutting fluids at six o'clock is working on a small fraction of the problem. None of them clear anything, which is why the dose climbs, why a second drug shows up, and why the appointment eventually stops being about pills and starts being about a catheter, a TURP procedure, or retention you did not see coming. That escalation isn't bad luck. Tamsulosin costs about nine dollars for ninety capsules. The surgical market waiting behind it is worth seventeen billion by 2030 and growing every year. Nobody in that chain earns a dollar from the pill. Everybody earns when it stops working. The pill was never the treatment. It's the waiting room. What actually clears a fibrin mesh is an enzyme. The one with the most human data behind it comes from natto, a fermented soybean the Japanese have eaten for a thousand years. A researcher named Sumi found it in 1980 when he dropped some onto a dish of artificial clot and watched it dissolve. It's called nattokinase. In a trial of 1,062 people, it measurably reduced arterial plaque and wall thickness. But clearing the vessels is only one of three things that have to happen, and this is where almost every product on the shelf fails. You have to clear the mesh. You have to calm the tissue that has already inflamed. And you have to keep the calcium that gets freed from walking straight back into the vessel wall. The formula I ended up on is the only one I found that does all three. Noverly Nattokinase, 10,000 FU. The enzyme for the mesh. Turmeric, pine bark extract, aged garlic and bromelain for the tissue — curcumin has a meta-analysis behind it across six studies and 697 men showing measurable improvement in urinary symptom scores, and pine bark has a study of 75 men aged 55 to 75 showing a 31% reduction in getting up at night and a 51% improvement in what's left in the bladder afterwards. And vitamin K2 as MK-7, which routes the freed calcium into bone instead of back into the artery wall. Every prostate supplement on the market does the second layer and ignores the first. Every nattokinase brand does the first and has never once said the word prostate. I need to be straight with you about two things, because I'd rather lose the sale. There is no trial on nattokinase and ejaculation. I am not telling you this brings anything back. What I'm telling you is that what stopped coming out was your body's alarm about your blood supply, and this is aimed at your blood supply. And do not stop your medication. Not over a Facebook post, not over anything I've written here. That conversation belongs to you and your doctor. This runs alongside it. When men get results, the way that conversation usually starts is their doctor bringing up the dose first. If you're on a blood thinner — warfarin, apixaban, rivaroxaban, or a daily aspirin — or you have surgery scheduled, this is not for you. I mean that. Give it three weeks before you expect anything and eight before you judge it. Count your bathroom trips tonight and write the number down. That's your baseline. In three weeks, count again. You were never given the choice between sleeping through the night and being a man. Someone made that trade on your behalf, and they made it on a false premise. → noverly.shop/products/nattokinase-10-000-fu-clinical-strength-heart-circulation-complex P.S. — My father counted. He'd been writing down the number for years, which is very much like him. He went from four a night to one over about seven weeks. He has never once told me whether anything else changed, and I have never once asked him. Some things a man gets to keep to himself. The point is that he got to choose.
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Discover The Power of Natto Today!
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