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If your urologist trained at a teaching hospital in the last 20 years, there is a reasonable chance I helped train the surgeon who is about to schedule you for HoLEP. And I need to tell you what I did not teach them — because I was not taught it either. I have taught HoLEP technique to more than 200 urology residents over the last two decades at a large academic medical center in the northeast. I helped design the surgical skills curriculum. I proctored the first fifty HoLEP procedures of residents who are now attending urologists in community hospitals in twenty-three states. I am Dr. James Kotler. Board-certified urologist. Thirty years in practice. Attending urologic surgeon at an academic teaching hospital. Fellowship-trained in endourology. I still perform HoLEP and TURP when it is truly necessary — kidney damage, complete urinary blockage, men in their eighties whose prostates have grown for decades unchecked. What I see every week now, in my second-opinion clinic, is men in their sixties being scheduled for HoLEP by community urologists who trained under me and who are following the surgical algorithm I taught them. Men whose sexual function was already fragile and is about to be finished by a procedure they were told preserved it. Men who walk out of my office three months later with retrograde ejaculation, weaker erections, and a wife who quietly stopped initiating. I helped train the doctors who are scheduling them. I did not teach the residents to look at the root cause of BPH because I was not taught to look at it myself. Every HoLEP brochure says the same three things. Preserves erectile function in most patients. Minimally invasive. Gold standard for prostates over eighty grams. What every HoLEP brochure does not say. Seventy-five to ninety percent of men experience retrograde ejaculation after the procedure. Permanently. Your ejaculate goes backwards into your bladder instead of forwards. Most men describe this as the moment their sex life changed forever, regardless of whether they could still get hard. And the erectile preservation rate. Better than TURP, yes. But independent long-term studies show ten to twenty percent of HoLEP patients develop new-onset erectile dysfunction within twenty-four months. The kind that does not come back. Your urologist is not lying to you. He is repeating the algorithm I taught him. And the algorithm is incomplete. I have been prescribing tamsulosin for 25 years to delay men from reaching the HoLEP conversation. Thousands of men. Every one of them told the same thing — this will help with your symptoms while we watch the prostate. And I have seen what happens three, four, five years later when the tamsulosin stops being enough and the HoLEP conversation finally arrives. The 63-year-old who took tamsulosin faithfully for three years, never missed a dose, then his urologist used the phrase we should talk about a procedure. Within six months he had HoLEP. Eighteen months later he sat in my second-opinion clinic because his ejaculate had been going backwards ever since, his erections had quietly weakened, and his wife had stopped reaching for him at night. The 67-year-old. Six years on Flomax. Then the maximum dose stopped working. His community urologist scheduled HoLEP within four weeks. The retrograde ejaculation hit him on the third night after the catheter came out. His urologist told him it was a minor trade-off. His wife found out from him crying in the kitchen six months later. The 71-year-old. His urologist actually laughed when he asked if there was any way to avoid the procedure. He scheduled HoLEP because he was exhausted from waking up four times a night. Sexual function had already been fading. HoLEP took what remained. I see these men in follow-up because they seek me out. They looked up who trained the surgeon who operated on them. My name comes up because I was on the CV. They come to me hoping I can undo what my former resident did. I cannot undo it. That is why I am writing this — so the next man does not get on the table. When my own prostate symptoms started at 55, I knew exactly where I was heading. Waking up three, four times a night. Stop-start stream. The constant feeling of never fully emptying. And erections that were taking longer to achieve, getting weaker, sometimes failing halfway through. My colleague at the university looked at my symptoms and said what urologists say to every patient. Standard protocol. Start with tamsulosin 0.4mg. Should help with the flow. We will watch the prostate size. We will watch the prostate size. I know what that phrase means. I have said it to hundreds of patients. It means the HoLEP conversation is already on the timeline. Five years. Maybe seven if I was lucky. I am a urologist and I have trained other urologists. I know my own prostate trajectory. Tamsulosin would buy me time. But the prostate would keep growing. The erectile function would keep declining. And one Tuesday morning, in five or seven years, I would be sitting on the other side of the same operating table where I had trained residents on 400 men before me, signing the same consent form. I started researching what was actually happening inside my prostate and my erectile tissue. What I found made me furious that I had been teaching HoLEP technique for twenty years without teaching what caused BPH in the first place. Your prostate is not enlarging randomly. And your erections are not failing randomly. They are the same problem. Same root causes. Destroying both simultaneously and walking you toward the HoLEP table together. The first is DHT overproduction. After 40, your body converts testosterone into DHT at an accelerating rate. DHT acts like fertilizer on prostate tissue, forcing it to grow larger every year without stopping. The bigger your prostate gets, the more aggressively your urologist will recommend HoLEP. But DHT conversion also disrupts the hormonal balance needed for healthy erectile function. The second is vascular dysfunction. Poor blood flow to the prostate creates chronic hypoxia. When prostate cells cannot get enough oxygen, they trigger compensatory inflammation. The prostate swells. Presses harder against your urethra. That same vascular dysfunction is destroying your erectile tissue. Your penis needs massive blood flow to achieve erections. When the pelvic vessels are damaged, constricted, unable to dilate, erections become weak, unreliable, then impossible. The third is chronic NF-kappa-B-driven inflammation. This inflammatory cascade causes tissue proliferation and fibrosis in your prostate. Year after year the tissue thickens and the urethra narrows toward the point where HoLEP becomes the only conversation. But that same inflammation is destroying your ability to produce nitric oxide. That is the gas that causes erections. Without it, you cannot get hard. Period. Tamsulosin addresses none of these. Zero. It relaxes the muscles around your prostate for ten to twelve hours so you can urinate. Then it wears off. The DHT is still forcing prostate growth. The inflammation is still choking your urethra. The vascular dysfunction is still starving both organs of oxygen. HoLEP does not address the root causes either. It laser-removes the prostate tissue that has already grown. The DHT keeps converting. The inflammation keeps churning. The vascular dysfunction keeps destroying erectile capacity. That is why so many post-HoLEP men show up in my clinic two years later with new or worsening sexual dysfunction. The surgery fixed the plumbing. It did not touch the engine. That is not treatment. That is a business model that ends on an operating table I helped train the surgeon to run. In February of last year, at 1:47 AM after a long resident evaluation session, I was reviewing urology and vascular research on my laptop at the kitchen table. My own symptoms had gotten worse over the winter. Four trips to the bathroom the night before. Erections that were now unreliable more often than reliable. I was exhausted and getting angry. I was going through studies on BPH pathophysiology, vascular function, and endothelial biology. Not looking for anything specific. Just determined to find something the surgical algorithm was missing before I ended up on the table I had put other men on for a decade. Then I found it. A 2024 study in Naunyn-Schmiedeberg's Archives of Pharmacology. Capsaicin from cayenne pepper activates TRPV1 receptors in endothelial cells, prostate tissue, and erectile tissue. TRPV1 activation triggers sustained nitric oxide release. Nitric oxide causes vasodilation and inhibits hypoxia-driven tissue proliferation. The exact mechanism for all three BPH pathways and erectile restoration. Capsaicin treatment reduced prostate weight by 31 percent. Decreased inflammatory markers (NF-kappa-B, COX-2). Improved endothelial nitric oxide synthase expression. Inhibited 5-alpha reductase activity simultaneously. And restored erectile function by re-establishing nitric oxide production in penile tissue. All three root causes. Both problems. One compound. Multi-pathway efficacy the pharmaceutical algorithm could not match because tamsulosin only targets one pathway and HoLEP does not target the mechanism at all. I sat at the kitchen table looking at what I had been missing for 30 years of practice and 20 years of training other urologists. The mechanism that would have kept hundreds of my patients — and hundreds of the patients of the residents I trained — off the HoLEP table if I had known about it ten years earlier. Capsaicin activates TRPV1 receptors throughout the pelvic vascular system. Continuous nitric oxide release. Natural sustained vasodilation in both prostate and penile tissue. Oxygen-starved tissue gets fresh blood. When the cells can breathe again, the inflammation calms. The prostate swelling reduces. The squeeze on the urethra loosens. The erectile tissue starts producing nitric oxide again. That is what allows natural erections. And it inhibits 5-alpha reductase — regulating DHT conversion at the source so the prostate stops growing toward the HoLEP threshold. I searched for pharmaceutical-grade capsaicin that night. Not cayenne pepper capsules from the health food store. Real capsaicin. Dissolved in oil for bioavailability. At the dose the research supports. I found Aurivita Capsaicin Power. 3mg capsaicin per serving. Pre-dissolved in cold-pressed avocado oil — the exact delivery system required for absorption. With BioPerine to block liver breakdown. With K2 to prevent calcium deposition during vascular repair. With beetroot extract for additional nitric oxide support. Not a trace amount to justify a label. The real therapeutic dose. I ordered it that night. I began taking it the next morning. Three softgels with dinner. Day 12. I woke up at 5:52 AM. Not because I needed to urinate. Because sunlight was coming through the window. I lay there for a moment just processing it. I had slept almost six hours straight for the first time in more than two years. Week 3. Saturday morning. Standing over the toilet. A strong steady stream. No hesitation. No stopping and starting. No pushing. I stood there genuinely amazed. I had forgotten what normal urination felt like. Week 4. Morning wood. First time in over a year. I did not think about it. Did not try to make it happen. Just woke up with a full erection pressing against the sheets. Month 2. I ordered my own follow-up urological testing through a colleague at another institution. Flow rate up from 8 ml/s to 17 ml/s. Residual volume down from 140ml to 25ml. Prostate volume down 18 percent on ultrasound. Erectile function fully restored. My department chair asked me at faculty rounds what I was doing. He said my energy was different. I told him. He asked me to write it up. I started recommending Aurivita to patients coming through my second-opinion clinic. Men whose community urologists — the ones I had trained — had already scheduled them for HoLEP. Robert. 68. HoLEP scheduled for the following month. His urologist was one of my former residents. Robert had been on tamsulosin for six years and stopped responding. Erectile function already unreliable. Two weeks on Aurivita — nothing. Week 3, slept until 5:30 AM without a trip to the bathroom. Week 4, morning erection returned. By week 8 his stream had doubled in force and his erectile function was consistent. He called his urologist and cancelled the HoLEP. His urologist called me demanding to know what I had said to him. I told him. He hung up. David. 71. HoLEP consultation the week after we met. Maximum-dose tamsulosin, still getting up four to five times a night. Erections weak and unreliable. Eight weeks on Aurivita his flow rate had doubled. Morning wood returned. Cancelled the consultation. His GP asked him to write down what he was taking. Michael. 65. Six years on Flomax. His urologist had already booked his pre-op assessment. Michael's older brother had permanent retrograde ejaculation from a TURP in 2018, and Michael had watched what it did to his brother's marriage. He was terrified. We worked together to taper the tamsulosin slowly while taking Aurivita. Now getting up once a night. Sometimes not at all. Erections came back at full strength. He cancelled the HoLEP and kept his ejaculation. In thirty years I had never seen results like that without surgical intervention. And in the twenty years I had been training residents on HoLEP, none of us had considered that BPH was reversible through vascular repair. I am not telling you this because I am against HoLEP. It manages mechanical obstruction. It works as designed for what it is designed to do. For an 84-year-old with complete urinary retention and kidney compromise, HoLEP saves lives. I will continue performing it and continue training residents on it. But HoLEP does not stop DHT overproduction. It does not restore blood flow to oxygen-starved prostate and erectile tissue. It does not calm the NF-kappa-B inflammatory cascade. It does not activate the TRPV1 receptors that trigger your body's natural healing mechanisms in both organs. It does not restore nitric oxide production. It does not bring back erections. It surgically removes prostate tissue while all three root causes keep destroying your sexual function underneath. And I see what happens to the men who go through with it when they did not have to. A 2023 review of HoLEP outcomes across 14 studies found seventy-five to ninety percent of patients develop retrograde ejaculation post-procedure. Permanently. New-onset erectile dysfunction within 24 months in ten to twenty percent. Stress urinary incontinence in one to twelve percent depending on the surgeon's experience. Urethral stricture in three to five percent. Bladder neck contracture in one to three percent. The marketing material focuses on what HoLEP preserves. The post-operative reality is what it takes. Your symptoms right now — the night-time trips, the weak stream, the urgency, the erections getting weaker, the consultations your urologist keeps scheduling — they are your warning system. The warning before the table. And you have two choices. Schedule the HoLEP your urologist wants to perform and accept the seventy-five to ninety percent chance of retrograde ejaculation and the ten to twenty percent chance of new-onset ED you will spend the rest of your life with. Or activate TRPV1 and address all three root causes in both organs while there is still time to keep your prostate, your ejaculation, and your erectile function. I think about what I almost did. I almost spent five more years on tamsulosin watching my prostate grow toward the HoLEP threshold I had been training residents to cross for twenty years. I almost became the patient I had been teaching my residents to operate on, wishing someone had told me earlier that the procedure I taught for a living was the one I should have done everything possible to avoid. Because once you sign the HoLEP consent form, the trade-offs are permanent. Retrograde ejaculation in seventy-five to ninety percent of patients. New-onset ED in ten to twenty percent. Stress incontinence in some. Urethral stricture in others. The preservation of erectile function the brochure promises is a statistical average, not a guarantee. For one in five men, it is a lie they only discover after the catheter comes out. You do not get a warning before that point. Your symptoms right now are the warning. Use them. Aurivita Capsaicin Power costs $54 for 60 days. Less than $1 a day. Compare that to $15,000 to $25,000 for self-pay HoLEP in the US, or thousands more once you factor in pre-op assessments, anesthesia, hospital stay, follow-up consultations, and lost workdays. Compare that to the cost of antegrade ejaculation, which has no price tag because you cannot buy it back once it is gone. It comes with a 120-day money-back guarantee. Take it for three full months — two full bags. If your stream is not stronger, if you are not sleeping better, if your morning erections are not returning, if your prostate symptoms are not improving, send the bags back — even empty — for a full refund. No questions asked. I have never seen a pharmaceutical company offer that. I have never seen a HoLEP procedure come with a money-back guarantee. And I have never seen a treatment that protects your prostate AND your sexual function at the same time. That is how confident I am in what TRPV1 activation actually does. Save your prostate and your sex life now, before you sign the consent form. https://aurivita.co/products/capsaicin-power-ed Dr. James Kotler, MD, FACS Board-Certified Urologist, 30 Years in Practice Attending Urologic Surgeon, Academic Teaching Hospital P.S. Give it 60 to 90 days. See the difference for yourself. You have nothing to lose except $54 and 8 to 12 weeks. The upside is keeping your prostate intact, keeping your antegrade ejaculation, keeping your erectile function, and walking away from the HoLEP consultation my former resident has already pencilled in for you. P.P.S. If your urologist trained in the northeast between 2005 and 2018, there is a real chance he was in one of my proctored cases. If he tells you HoLEP preserves sexual function, know that I was the one who taught him that phrase. And know that I now understand — after 30 years and my own recovery — how incomplete that phrase always was.
I Trained 200 Urology Residents On HoLEP. I Never Taught What Caused BPH.
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